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| A small proportion of these people will develop hemochromatosis symptoms. Inheriting two copies of H63D is very rare, but it does happen. Some say people with two copies of the H63D mutation are at risk of developing hemochromatosis, while others disagree. Secondary hemochromatosis This type of hemochromatosis occurs as a result of another condition or circumstance (the patient does not have C282Y or H63D mutations). Examples include: A blood disorder, such as thalassemia. Chronic liver disease, such as chronic hepatitis C infection, alcoholic liver disease, or non-alcoholic steatohepatitis. Some types of anemia which require blood transfusion. Rare inherited diseases that affect red blood cells, including atransferrinemia or aceruloplasminaemia.
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Drinking beer that has been brewed in iron containers (African iron overload). Oral iron pills or iron injections, with or without very high vitamin C intake. Juvenile hemochromatosis In juvenile hemochromatosis iron build up starts much earlier in life and symptoms appear between 25 and 30 years of age. The patient may develop diabetes and have problems with sexual development. Left untreated juvenile hemochromatosis can be fatal. Juvenile hemochromatosis is an inherited disease caused by a mutation in a gene called hemojuvelin (the HFE gene is not involved). Neonatal hemochromatosis Iron accumulates in the infant's liver so fast that he/she is either stillborn or dies not long after birth.
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